Human Endogenous Retrovirus Type K promotes proliferation of Merlin-negative schwannoma and meningioma which can be inhibited by anti-retroviral and anti-TEAD drugs

CO Hanemann; E Maze; B Agit; S Reeves; D Hilton; DB Parkinson; L Laraba; E Ercolano; K Kurian; R Belshaw; S Ammoun

doi:10.14791/btrt.2022.10.f-1131

Human Endogenous Retrovirus Type K promotes proliferation of Merlin-negative schwannoma and meningioma which can be inhibited by anti-retroviral and anti-TEAD drugs

CO Hanemann
, E Maze
, B Agit
, S Reeves
, D Hilton
, DB Parkinson
, L Laraba
, E Ercolano
, K Kurian
, R Belshaw
, S Ammoun

Peninsula Medical School

Research output: Contribution to conference › Conference paper (not formally published) › peer-review

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Original language	English
DOIs	https://doi.org/10.14791/btrt.2022.10.f-1131
Publication status	Published - 2022

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10.14791/btrt.2022.10.f-1131

Experimental and Molecular Therapeutics Abstract AACR meeting 2021

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title = "Human Endogenous Retrovirus Type K promotes proliferation of Merlin-negative schwannoma and meningioma which can be inhibited by anti-retroviral and anti-TEAD drugs",

author = "CO Hanemann and E Maze and B Agit and S Reeves and D Hilton and DB Parkinson and L Laraba and E Ercolano and K Kurian and R Belshaw and S Ammoun",

year = "2022",

doi = "10.14791/btrt.2022.10.f-1131",

language = "English",

}

TY - CONF

T1 - Human Endogenous Retrovirus Type K promotes proliferation of Merlin-negative schwannoma and meningioma which can be inhibited by anti-retroviral and anti-TEAD drugs

AU - Hanemann, CO

AU - Maze, E

AU - Agit, B

AU - Reeves, S

AU - Hilton, D

AU - Parkinson, DB

AU - Laraba, L

AU - Ercolano, E

AU - Kurian, K

AU - Belshaw, R

AU - Ammoun, S

PY - 2022

Y1 - 2022

UR - https://pearl.plymouth.ac.uk/pms-research/393

U2 - 10.14791/btrt.2022.10.f-1131

DO - 10.14791/btrt.2022.10.f-1131

M3 - Conference paper (not formally published)

ER -

Human Endogenous Retrovirus Type K promotes proliferation of Merlin-negative schwannoma and meningioma which can be inhibited by anti-retroviral and anti-TEAD drugs

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