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Endogenous GABA attenuates CNS white matter dysfunction following anoxia.
R Fern
, SG Waxman, BR Ransom
Faculty of Health
Research output
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Contribution to journal
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Article
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peer-review
Overview
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Dive into the research topics of 'Endogenous GABA attenuates CNS white matter dysfunction following anoxia.'. Together they form a unique fingerprint.
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Agricultural and Biological Sciences
Hypoxemia
100%
Protein Kinase C
83%
Action Potential
50%
G Protein
50%
Receptor
50%
Eicosanoid Receptor
50%
Agonist
33%
Protective Effect
16%
Optic Nerve
16%
Staurosporine
16%
Nerve Fiber
16%
Myristic Acid
16%
Immunology and Microbiology
White Matter
100%
Action Potential
100%
Agonist
66%
Nerve Fiber
33%
Optic Nerve
33%
Receptor Blocking
33%
Receptor down Regulation
33%
Pertussis Toxin
33%
Pharmacology, Toxicology and Pharmaceutical Science
4 Aminobutyric Acid
100%
Anoxia
100%
Protein Kinase C
35%
4 Aminobutyric Acid B Receptor
28%
Guanine Nucleotide Binding Protein
21%
Injury
14%
Baclofen
14%
Absence
7%
Desensitization
7%
Pertussis Toxin
7%
Axonal Injury
7%
Staurosporine
7%
Myristic Acid
7%
4 Aminobutyric Acid A Receptor Stimulating Agent
7%
1 (5 Isoquinolinesulfonyl) 2 Methylpiperazine
7%
Bicuculline
7%
Nipecotic Acid
7%
4 Aminobutyric Acid B Receptor Blocking Agent
7%
Neuroscience
Gamma-Aminobutyric Acid
100%
Protein Kinase C
31%
4 Aminobutyric Acid B Receptor
25%
G Protein
18%
Action Potential
18%
Agonist
12%
Baclofen
12%
Nerve Fiber
6%
Myristic Acid
6%
Receptor down Regulation
6%
Optic Nerve
6%
Staurosporine
6%
Bicuculline
6%
Epileptic Absence
6%
Pertussis Toxin
6%