Differential compaction due to the irregular topology of a diagenetic reaction boundary: A new mechanism for the formation of polygonal faults

We propose a new mechanism for the formation of some polygonal fault arrays. Seismically imaged opal-A (biogenic silica) to opal-CT (cristobalite and tridymite) diagenetic boundaries from two regions offshore of Norway have developed regular wavelength patterns. The pattern consists of cell-shaped elevations that are 200-2600m wide and up to 200m high, separated by troughs. The cells represent regions that undergo diagenesis at shallower burial depths, earlier than adjacent areas. The chemical change leads to mechanical compaction and porosity reduction; therefore subsidence occurs above the cells in the overburden. Roughly circular depressions form above the cells, and a network of folds form above inter-cell areas. Networks of normal faults form on the crests and margins of the folds as a result of flexure during the folding. The progressive lateral growth of the cells causes the depressions to widen and intervening folds to narrow resulting in new differential compaction-induced faults to form with variable strike orientations. Lateral and vertical growth of cells leads to cells conjoining and the re-establishment of a uniform planar reaction boundary. This novel but simple mechanism can explain some polygonal fault arrays that form above opal-A to opal-CT reaction boundaries and in these settings the mechanism should be considered in addition to syneresis, density inversion or low coefficients of residual friction which are the most commonly cited drivers for polygonal fault systems.