Δ9-THC is protective in a cell culture model of Parkinson's disease through a PPARγ mediated mechanism resulting in increased expression of PGC1-α

ML Zeissler; O Hanemann; JP Zajicek; CB Carroll

Δ9-THC is protective in a cell culture model of Parkinson's disease through a PPARγ mediated mechanism resulting in increased expression of PGC1-α

ML Zeissler
, O Hanemann
, JP Zajicek
, CB Carroll

Peninsula Medical School

Research output: Contribution to journal › Conference proceedings published in a journal › peer-review

Original language	English
Pages (from-to)	S39-S39
Number of pages	0
Journal	Movement Disorders
Volume	27
Issue number	0
Publication status	Published - 2012

Cite this

@article{86072328a4104a369889e11d048c3192,

title = "Δ9-THC is protective in a cell culture model of Parkinson's disease through a PPARγ mediated mechanism resulting in increased expression of PGC1-α",

author = "ML Zeissler and O Hanemann and JP Zajicek and CB Carroll",

year = "2012",

language = "English",

volume = "27",

pages = "S39--S39",

journal = "Movement Disorders",

issn = "0885-3185",

publisher = "John Wiley \& Sons Inc.",

number = "0",

}

Δ9-THC is protective in a cell culture model of Parkinson's disease through a PPARγ mediated mechanism resulting in increased expression of PGC1-α. / Zeissler, ML; Hanemann, O; Zajicek, JP et al.
In: Movement Disorders, Vol. 27, No. 0, 2012, p. S39-S39.

Research output: Contribution to journal › Conference proceedings published in a journal › peer-review

TY - JOUR

T1 - Δ9-THC is protective in a cell culture model of Parkinson's disease through a PPARγ mediated mechanism resulting in increased expression of PGC1-α

AU - Zeissler, ML

AU - Hanemann, O

AU - Zajicek, JP

AU - Carroll, CB

PY - 2012

Y1 - 2012

M3 - Conference proceedings published in a journal

SN - 0885-3185

VL - 27

SP - S39-S39

JO - Movement Disorders

JF - Movement Disorders

IS - 0

ER -

Δ<SUP>9</SUP>-THC is protective in a cell culture model of Parkinson's disease through a PPARγ mediated mechanism resulting in increased expression of PGC1-α

Cite this